Hepatoprotective action of schisandrin B against carbon tetrachloride toxicity was mediated by both enhancement of mitochondrial glutathione antioxidant status and induction of heat shock proteins in mice.

Kam Ming Ko; Man Ho Tang

Hepatoprotective action of schisandrin B against carbon tetrachloride toxicity was mediated by both enhancement of mitochondrial glutathione antioxidant status and induction of heat shock proteins in mice.

The Hong Kong University of Science and Technology

Research output: Contribution to conference › Conference Paper

Original language	English
Publication status	Published - 2002
Event	2nd International Symposium on Antioxidants in Nutrition & Therapy: Mechanisms in Physiology-Pathology-Pharmacology, Society for Free Radical Research, Bali, Indonesia - Duration: 1 Jan 2002 → 1 Jan 2002

Conference

Conference	2nd International Symposium on Antioxidants in Nutrition & Therapy: Mechanisms in Physiology-Pathology-Pharmacology, Society for Free Radical Research, Bali, Indonesia
Period	1/01/02 → 1/01/02

Access to Document

https://hdl.handle.net/1783.1/13048

Cite this

Ko, K. M., & Tang, M. H. (2002). Hepatoprotective action of schisandrin B against carbon tetrachloride toxicity was mediated by both enhancement of mitochondrial glutathione antioxidant status and induction of heat shock proteins in mice.. Paper presented at 2nd International Symposium on Antioxidants in Nutrition & Therapy: Mechanisms in Physiology-Pathology-Pharmacology, Society for Free Radical Research, Bali, Indonesia. https://hdl.handle.net/1783.1/13048

Ko, Kam Ming ; Tang, Man Ho. / Hepatoprotective action of schisandrin B against carbon tetrachloride toxicity was mediated by both enhancement of mitochondrial glutathione antioxidant status and induction of heat shock proteins in mice. Paper presented at 2nd International Symposium on Antioxidants in Nutrition & Therapy: Mechanisms in Physiology-Pathology-Pharmacology, Society for Free Radical Research, Bali, Indonesia.

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title = "Hepatoprotective action of schisandrin B against carbon tetrachloride toxicity was mediated by both enhancement of mitochondrial glutathione antioxidant status and induction of heat shock proteins in mice.",

author = "Ko, \{Kam Ming\} and Tang, \{Man Ho\}",

year = "2002",

language = "English",

note = "2nd International Symposium on Antioxidants in Nutrition \& Therapy: Mechanisms in Physiology-Pathology-Pharmacology, Society for Free Radical Research, Bali, Indonesia ; Conference date: 01-01-2002 Through 01-01-2002",

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Ko, KM & Tang, MH 2002, 'Hepatoprotective action of schisandrin B against carbon tetrachloride toxicity was mediated by both enhancement of mitochondrial glutathione antioxidant status and induction of heat shock proteins in mice.', Paper presented at 2nd International Symposium on Antioxidants in Nutrition & Therapy: Mechanisms in Physiology-Pathology-Pharmacology, Society for Free Radical Research, Bali, Indonesia, 1/01/02 - 1/01/02. <https://hdl.handle.net/1783.1/13048>

Hepatoprotective action of schisandrin B against carbon tetrachloride toxicity was mediated by both enhancement of mitochondrial glutathione antioxidant status and induction of heat shock proteins in mice. / Ko, Kam Ming; Tang, Man Ho.
2002. Paper presented at 2nd International Symposium on Antioxidants in Nutrition & Therapy: Mechanisms in Physiology-Pathology-Pharmacology, Society for Free Radical Research, Bali, Indonesia.

Research output: Contribution to conference › Conference Paper

TY - CONF

T1 - Hepatoprotective action of schisandrin B against carbon tetrachloride toxicity was mediated by both enhancement of mitochondrial glutathione antioxidant status and induction of heat shock proteins in mice.

AU - Ko, Kam Ming

AU - Tang, Man Ho

PY - 2002

Y1 - 2002

M3 - Conference Paper

T2 - 2nd International Symposium on Antioxidants in Nutrition & Therapy: Mechanisms in Physiology-Pathology-Pharmacology, Society for Free Radical Research, Bali, Indonesia

Y2 - 1 January 2002 through 1 January 2002

ER -

Ko KM, Tang MH. Hepatoprotective action of schisandrin B against carbon tetrachloride toxicity was mediated by both enhancement of mitochondrial glutathione antioxidant status and induction of heat shock proteins in mice.. 2002. Paper presented at 2nd International Symposium on Antioxidants in Nutrition & Therapy: Mechanisms in Physiology-Pathology-Pharmacology, Society for Free Radical Research, Bali, Indonesia.